Side effects of using viagra at a young age

In humans, Viagra has no effect on bleeding time when taken alone or with aspirin.Hearing: Cases of sudden decrease or loss of hearing have been reported postmarketing in temporal association with the use of PDE5 inhibitors, including Viagra.In healthy male volunteers, there was no evidence of a clinically significant effect of azithromycin (500 mg daily for 3 days) on the systemic exposure of sildenafil or its major circulating metabolite.Viagra (50 mg) did not potentiate the increase in bleeding time caused by aspirin (150 mg).Effects Of Viagra At A Young Age plays a vital role in inspiring consumers to reach for your products.

Physicians should advise patients to stop use of all phosphodiesterase type 5 (PDE5) inhibitors, including Viagra, and seek medical attention in the event of a sudden loss of vision in one or both eyes.Use caution when co-administering alpha-blockers with Viagra because of potential additive blood pressure-lowering effects.There is a potential for cardiac risk of sexual activity in patients with preexisting cardiovascular disease.

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The major circulating metabolite results from N-desmethylation of sildenafil, and is itself further metabolized.There have been postmarketing reports of bleeding events in patients who have taken Viagra.If you have an erection that lasts more than 4 hours, get medical help right away.Exhaustive RAND Corporation review of vaccine side effects finds strong.The time course of effect was examined in one study, showing an effect for up to 4 hours but the response was diminished compared to 2 hours.

In a study of healthy male volunteers, sildenafil (100 mg) did not affect the steady state pharmacokinetics of the HIV protease inhibitors, saquinavir and ritonavir, both of which are CYP3A4 substrates.Body as a Whole: face edema, photosensitivity reaction, shock, asthenia, pain, chills, accidental fall, abdominal pain, allergic reaction, chest pain, accidental injury.

Prolonged erection greater than 4 hours and priapism (painful erections greater than 6 hours in duration) have been reported infrequently since market approval of Viagra.One-third to one-half of the subjects in these studies reported successful intercourse at least once during a 4-week, treatment-free run-in period.I could feel my penis get erect within 15 minutes, getting harder than.Causes Of Erectile Dysfunction At Young Age Finasteride Erectile Dysfunction with Male Enlargement. trying to draw in public attention towards Viagra side-effects.Stepwise increase in alpha-blocker dose may be associated with further lowering of blood pressure when taking a PDE5 inhibitor.In addition to human corpus cavernosum smooth muscle, PDE5 is also found in other tissues including platelets, vascular and visceral smooth muscle, and skeletal muscle, brain, heart, liver, kidney, lung, pancreas, prostate, bladder, testis, and seminal vesicle.Skin and Appendages: urticaria, herpes simplex, pruritus, sweating, skin ulcer, contact dermatitis, exfoliative dermatitis.Therefore, PDE5 inhibitors, including Viagra, should be used with caution in these patients and only when the anticipated benefits outweigh the risks.Subjects who had successfully completed the previous doxazosin interaction study (using Viagra 50 mg), including no significant hemodynamic adverse events, were allowed to skip dose period 1.

Following at least 14 consecutive days of doxazosin, Viagra 50 mg or matching placebo was administered simultaneously with doxazosin 4 mg (17 subjects) or with doxazosin 8 mg (3 subjects).

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Sildenafil has no direct relaxant effect on isolated human corpus cavernosum.Blood pressure was measured immediately pre-dose and at 15, 30, 45 minutes, and 1, 1.5, 2, 2.5, 3, 4, 6 and 8 hours after Viagra or matching placebo.Others were reported to have occurred hours to days after the use of Viagra and sexual activity.The primary measure in the principal studies was a sexual function questionnaire (the International Index of Erectile Function - IIEF) administered during a 4-week treatment-free run-in period, at baseline, at follow-up visits, and at the end of double-blind, placebo-controlled, at-home treatment.The safety and efficacy of combinations of Viagra with other PDE5 Inhibitors, including REVATIO or other pulmonary arterial hypertension (PAH) treatments containing sildenafil, or other treatments for erectile dysfunction have not been studied.

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When Viagra was taken as recommended (on an as-needed basis) in flexible-dose, placebo-controlled clinical trials of two to twenty-six weeks duration, patients took Viagra at least once weekly, and the following adverse reactions were reported.Sixty-three percent, 74%, and 82% of the patients on 25 mg, 50 mg and 100 mg of Viagra, respectively, reported an improvement in their erections, compared to 24% on placebo.Outliers were defined as subjects with a standing systolic blood pressure of 30 mmHg at one or more timepoints.In the event of an erection that persists longer than 4 hours, the patient should seek immediate medical assistance.

Metabolic and Nutritional: thirst, edema, gout, unstable diabetes, hyperglycemia, peripheral edema, hyperuricemia, hypoglycemic reaction, hypernatremia.These events have been chosen for inclusion either due to their seriousness, reporting frequency, lack of clear alternative causation, or a combination of these factors.PDE5 inhibitors, including Viagra, may potentiate the hypotensive effects of GC stimulators.

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Following a review of the data from these first 4 subjects (details provided below), the Viagra dose was reduced to 25 mg.Serious cardiovascular, cerebrovascular, and vascular events, including myocardial infarction, sudden cardiac death, ventricular arrhythmia, cerebrovascular hemorrhage, transient ischemic attack, hypertension, subarachnoid and intracerebral hemorrhages, and pulmonary hemorrhage have been reported post-marketing in temporal association with the use of Viagra.Sildenafil is also marketed as REVATIO for the treatment of PAH.Sildenafil was negative in in vitro bacterial and Chinese hamster ovary cell assays to detect mutagenicity, and in vitro human lymphocytes and in vivo mouse micronucleus assays to detect clastogenicity.Patients with cardiac failure or coronary artery disease causing unstable angina.Because these reactions are reported voluntarily from a population of uncertain size, it is not always possible to reliably estimate their frequency or establish a causal relationship to drug exposure.